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Compromised intestinal epithelial barrier due to intestinal epithelial-specific loss of JAM-A induces adaptive immune compensation that protects from colitis The findings in mice manipulated for MLCK or occludin expression differed from those obtained in mice lacking the JAM-A (encoded by F11r)

doi: 10.1111/joim.12564 564 MitchellWM

Extrapolating from other GLP-1 medications, long-term treatment appears necessary for maintaining benefits in most people

In addition, TNF- increases myosin light chain kinase (MLCK) phosphorylation [101], which may alter paracellular permeability through its association with actin and myosin (Fig